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Author(s): 

MUTSUSAKA T. | JIN Y.T.

Issue Info: 
  • Year: 

    2002
  • Volume: 

    54
  • Issue: 

    -
  • Pages: 

    568-575
Measures: 
  • Citations: 

    1
  • Views: 

    99
  • Downloads: 

    0
Keywords: 
Abstract: 

Yearly Impact: مرکز اطلاعات علمی Scientific Information Database (SID) - Trusted Source for Research and Academic Resources

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Issue Info: 
  • Year: 

    2007
  • Volume: 

    18
Measures: 
  • Views: 

    113
  • Downloads: 

    0
Keywords: 
Abstract: 

Introduction: To date, there is no report about protective effects of natural honey against ischemia/reperfusion injuries (esp. Infarct size) so that effects of short-term application of honey on ischemia/reperfusion-induced Infarct size were studied in isolated rat hearts.Methods: The isolated hearts were divided into three groups randomly (n=6 in each group) and mounted on a Langendorff apparatus at constant pressure. In control group, the hearts were perfused by a modified Krebs-Henseleit solution at stabilization, 30min regional ischemia and 120min reperfusion while in the test groups, they were perfused by enriched Krebs solution with natural honey (0.5 and 1%) 10min before to 10min after ischemia. At the end of reperfusion, Evans blue solution was infused to stain the non-ischemic area. Then the hearts were cut into slices and incubated by 1% Triphenyltetrazolium chloride (TTC) solution and fixed in formalin. The area of infracted tissue and risk zone were determined by a computerized planimetry.Results: The results demonstrated that short-term perfusion of isolated rat hearts with natural honey produces significant reduction in Infarct size. In the control group, Infarct size was 46.3±2.9%, however perfusion of natural honey (0.5 and 1%) reduced it to 17.9±5 and 22.6±7 (P<0.001 for both), respectively. The effect was not statistically significant between the test groups (P<0.05) In addition, statistical comparison between the control and test groups did not show significant difference in risk zone area.Conclusion: It seems the presence of antioxidant compounds such as phenolic and organic nonaromatic acids in natural honey may have important roles in Infarct size reduction.

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Journal: 

NEUROSURGICAL REVIEW

Issue Info: 
  • Year: 

    2000
  • Volume: 

    23
  • Issue: 

    -
  • Pages: 

    145-150
Measures: 
  • Citations: 

    1
  • Views: 

    179
  • Downloads: 

    0
Keywords: 
Abstract: 

Yearly Impact: مرکز اطلاعات علمی Scientific Information Database (SID) - Trusted Source for Research and Academic Resources

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Issue Info: 
  • Year: 

    2015
  • Volume: 

    22
Measures: 
  • Views: 

    175
  • Downloads: 

    64
Abstract: 

INTRODUCTION: ALTHOUGH REPERFUSION IS A USEFUL METHOD FOR THE SURVIVAL OF ISCHEMIC HEART, BUT IT HAS ADDITIONAL HARMFUL EFFECTS. VANILLIC ACID IS AN OXIDIZED FORM OF VANILLIN PRODUCED DURING THE CONVERSION OF VANILLIN TO FERULIC ACID AND HAS FREE RADICAL SCAVENGING, ANTIOXIDANT AND ANTI-INFLAMMATORY PROPERTIES. THIS STUDY WAS DESIGNED TO INVESTIGATE THE EFFECTS OF VANILLIC ACID ON HEMODYNAMIC PARAMETERS AND Infarct size IN ISCHEMIA-REPERFUSION OF ISOLATED RAT HEART.METHODS: FORTY MALE SPRAGUE DAWLEY RATS WERE RANDOMLY DIVIDED TO CONTROL AND THREE TREATMENT GROUPS. THREE DOSES OF VANILLIC ACID (5, 10 AND 20 MG/KG) WERE ADMINISTERED ORALLY TO RATS FOR 10 DAYS, THEN THE HEARTS ISOLATED AND WERE EXPOSED TO 30 MIN ISCHEMIA AND 1 HOUR REPERFUSION, USING LANGENDORFF APPARATUS. THE EFFECTS OF VANILLIC ACID, ON LV PRESSURE (LVP), LEFT VENTRICULAR DEVELOPED PRESSURE (LVDP), LV END DIASTOLIC PRESSURE (LVEDP), PEAK RATE OF RISE AND FALL OF LVP (±DP/DT), CORONARY FLOW (CF), RATE PRESSURE PRODUCT (RPP) AND Infarct size WERE EXAMINED. Infarct size WAS MEASURED AS PERCENTAGE RATIO OF THE Infarct AREA TO THE TOTAL AREA. THE SUCCESSFUL INDUCTION OF ISCHEMIA WAS DETERMINED BY ST ELEVATION ON THE ELECTROCARDIOGRAM.RESULTS: RATS ADMINISTERED VANILLIC ACID (10 AND 20 MG/KG), DISPLAYED IMPROVED RECOVERY OF LVEDP, RPP, LVDP, LVP AND ± DP/DT AS COMPARED TO CONTROL GROUP, SIGNIFICANTLY. THERE WAS ALSO SIGNIFICANT BENEFICIAL EFFECT OF THESE 2 DOSES TO REDUCE Infarct size.CONCLUSION: THE RESULTS SUGGEST THAT VANILLIC ACID CAN EFFECTIVELY IMPROVE VENTRICULAR FUNCTION AND REDUCE Infarct size IN ISCHEMIA-REPERFUSION OF ISOLATED RAT HEART.

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Issue Info: 
  • Year: 

    2007
  • Volume: 

    18
Measures: 
  • Views: 

    202
  • Downloads: 

    0
Keywords: 
Abstract: 

Introduction: To date, there is not reported any comparative study between the effects of Etomoxir and Ranolazine on ischemia/reperfusion injuries (esp. Infarct size), so that their effects on Infarct size in the ischemic isolated rat heart were studied and compared.Methods: Isolated rat hearts were divided into 3 groups randomly (n=6 in each group) and mounted on a non-recirculating Langendorff apparatus at constant pressure. In control group, the hearts were perfused by a modified Krebs-Henseleit solution at stabilization, 30min regional ischemia and 120min reperfusion while in the test groups; they were perfused by enriched Krebs solution with 1µM of Etomoxir or 20µM of Ranolazine during ischemia/reperfusion. At the end of reperfusion, Evans Blue solution was infused to stain the non-ischemic area. Then the heart was cut into slices and incubated by 1% TTZ solution and fixed in formalin. The area of infracted tissue and area at risk were determined by a computerized planimetry. Results: In the control group, Infarct size was 46.3±2.9%, while Etomoxir reduced Infarct size  to 20.9±5% (P>0.01) Perfusion of the hearts by Ranolazine enriched Krebs solution produced greater reduction in Infarct size (0.001>p, %3.6±16.4) The effect was not statistically significant between the test groups. Conclusion: It seems that Etomoxir (a fatty acid uptake inhibitor) and Ranolazine (a fatty acid oxidation inhibitor) likely by indirect increasing of glucose oxidation may improve reperfusion recovery of ischemic heart and reduce Infarct size. The results showed protective effects of Etomoxir and Ranolazine on Infarct size without any significant difference between the agents.

Yearly Impact:   مرکز اطلاعات علمی Scientific Information Database (SID) - Trusted Source for Research and Academic Resources

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Issue Info: 
  • Year: 

    2009
  • Volume: 

    19
Measures: 
  • Views: 

    142
  • Downloads: 

    0
Abstract: 

As no studies performed about pharmacologic effects of grape seed extract (GSE) on ischemia/reperfusion-induced injuries especially Infarct size, we performed this study to investigate effects of GSE on isolated rat hearts. Male SD rats were divided into four groups (n=8 in each group) and isolated hearts mounted on Langendorff apparatus under constant pressure and temperature. After stabilization (30 min), regional ischemia induced for 30 min followed by 120 min reperfusion. In the control group, the hearts were perfused with extract free Krebs-Henseleit (K/H) solution, while in the test groups; they were perfused 1, 10 and 100 mg/l of GSE enriched K/H solution from 15 min before ischemia to 120 min reperfusion. Then the hearts colored by evans blue and incubated by triphenyl tetrazolim chloride (TTZ). The area of infacted tissue was determined by computerized planimetry. The results show that perfusion of GSE produced significant reduction in Infarct size from 31±5% in control group to 12±3%, 16±6% (p<0.05) and 2±1% (p<0.01) in the test groups of GSE (1, 10 and 100 mg/l). The results can be attributed to the protective effects of proanthocyanidins to their ability to eliminatehydroxyl radicals. Also they may interact with intracellular calcium ions, leading to a reduction in reperfusion-induced calcium overload.

Yearly Impact:   مرکز اطلاعات علمی Scientific Information Database (SID) - Trusted Source for Research and Academic Resources

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Issue Info: 
  • Year: 

    2008
  • Volume: 

    -
  • Issue: 

    4
  • Pages: 

    1-11
Measures: 
  • Citations: 

    1
  • Views: 

    1308
  • Downloads: 

    0
Abstract: 

Objectives: In this study, prophylactic effects of natural honey as a pharmacologic preconditioning agent on ischemia/reperfusion (I/R) induced cardiac arrhythmias and Infarct size were investigated in isolated rat heart. Methods: Isolated rat hearts were mounted on a Langendorff apparatus then subjected to 30min regional ischemia followed by 120min reperfusion. In control group, the hearts were perfused by a modified Krebs-Henseleit solution throughout the experiment, however, in the test groups they were perfused by enriched Krebs solution with natural honey (0.25, 0.5 and 1%) 10min before to 10min after ischemia. The arrhythmias were analyzed based on the Lambeth conventions. The Infarct size was determined by Triphenyltetrazolium chloride and planimetry methods. Results: At the ischemia, the total number of ventricular ectopic beats (VEBs) in the control group was 667±116 while perfusion of natural honey (0.25, 0.5 and 1%) reduced it to 128±35 (p<0.01), 161±35 (p<0.01) and 303±94 (p<0.05), respectively. The number of ischemic ventricular tachycardia (VT) and the time spent in VT were significantly lowered by the same concentrations. Honey (0.25 and 0.5%) decreased the incidence of VT from 100% (control) to 13% (p<0.001) and 25% (p<0.01), respectively. During reperfusion phase, the number of VEBs and VT were significantly reduced by all used concentrations. In addition, honey (0.25%) significantly decreased the incidence and time spent for reversible ventricular fibrillation. Moreover, perfusion of honey (0.25, 0.5 and 1%) reduced Infarct size from 46.3±2.9% (control value) to 3.3±1.3, 9.2±1.9 and 11.7±2.2%, respectively (p<0.001 for all). Conclusion: The results of this study showed antiarrhythmic and anti-Infarct properties of natural honey as a preconditioning agent against I/R injuries in isolated rat heart. Probably, antioxidant activity of honey, scavenging of free radicals and presence of energy sources such as glucose may involve in these cardioprotective effects.

Yearly Impact: مرکز اطلاعات علمی Scientific Information Database (SID) - Trusted Source for Research and Academic Resources

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Issue Info: 
  • Year: 

    2013
  • Volume: 

    21
Measures: 
  • Views: 

    307
  • Downloads: 

    106
Abstract: 

INTRODUCTION AND AIM: SOME PREVIOUS STUDIES HAVE SHOWN THAT FRUCTOSE CAN PROTECT ISCHEMICREPERFUSED HEART AGAINST ISCHEMIA/REPERFUSION (I/R) INJURIES WHEREAS SOME OTHERS DEMONSTRATED THAT IT HAS A HARMFUL EFFECT IN SUCH CONDITION. IN THE PRESENT STUDY…

Yearly Impact:   مرکز اطلاعات علمی Scientific Information Database (SID) - Trusted Source for Research and Academic Resources

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Issue Info: 
  • Year: 

    2007
  • Volume: 

    -
  • Issue: 

    1
  • Pages: 

    23-28
Measures: 
  • Citations: 

    0
  • Views: 

    902
  • Downloads: 

    0
Abstract: 

Objectives: In this study, effects of ischemic postconditioning (IPC) and pharmacologic postconditioning (PPC) by using L-Carnitine (L- Car) on Infarct size in the ischemic-reperfused isolated rat heart were investigated and compared. Methods: Male rats were divided in five groups (control, IPC, and three PPC groups treated by L- Car) and were anesthetized by sodium pentobarbital (50 mg/kg-ip). Heart was removed and quickly mounted on a Langendorff apparatus and perfused by a modified Krebs- Henseleit (K/H) solution that was previously equilibrated with 95% O2–5% CO2. The hearts were subjected to 30 min regional ischemia followed by 120 min reperfusion. In the control and IPC groups, the hearts were perfused by normal K/H solution at stabilization, 30 min regional ischemia and 120 min reperfusion, while PPC groups were perfused by 0.5, 2.5 and 5mM of L-Car enriched K/H solution 10 min before and after reperfusion. At the end of reperfusion, Infarct size was determined by triphenyltetrazolium chloride method and computerized planimetry. Results: Infarct size was decreased significantly in both IPC and PPC groups versus control. In control group, Infarct size was 46.3±2.9 %, however, IPC reduced it to 22.6±1.5 % (p<0.001). Application of 0.5, 2, 5 and 5mM of Car-enriched K/H solution 10 min before and after reperfusion in the PPC groups, reduced the Infarct size from control group value to 41.8±4.0 (not significant), 28.1±2.0 (p<0.001) and 25.4±3.9 % (p<0.001), respectively. Except the effects of 0.5 mM L- Car, there was no significant difference between IPC and PPC groups on Infarct size reduction. Conclusion: Considering the results, it may be concluded that IPC and PPC (by L- Car) have protective effects against cardiac I/R injuries by reduction of Infarct size.

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Issue Info: 
  • Year: 

    2006
  • Volume: 

    9
  • Issue: 

    1
  • Pages: 

    29-35
Measures: 
  • Citations: 

    0
  • Views: 

    1816
  • Downloads: 

    0
Abstract: 

Background: The presence of C-reactive protein (CRP) during different stages of stroke had been shown in several studies. There is still no definite document about the correlation of CRP level and size of Infarct in stroke. We studied the correlation of the acute level of CRP with size of Infarct in stroke.Methods: A total of 90 consecutive patients with acute stroke admitted in Fatemeh Zahra University Hospital in Bushehr city were studied. Levels of CRP were measured at admission time and 48 hours later. sizes of Infarct and types of stroke were determined with Computerized Tomography scanning. The excluded patients were those with infection, stroke in brain stem, a delay more than 24 hours after attack of stroke and Transient Ischemic Attacks. CRP level was measured quantitatively using ELISA method.Results: No correlation between the first CRP levels and variables of age, size of Infarct and type of stroke was detected. size of Infarct was correlated with the second CRP (r=0.41, P<0.001) and the difference in CRP levels (r=0.45, P<0.001). The CRP difference was significant in ischemic, hemorrhagic and territory Infarcts (P<0.01). But there was no difference between the first and the second CRP in lacunar Infarcts.Conclusion: We found no correlation between the CRP levels of the 24 first hours after acute stroke with size of Infarct in stroke. But the increase of CRP levels which were measured in 48 hours after the stroke had correlation with size of Infarct irrespective of types of stroke. More studies could reveal the cause and effect of CRP in size of Infarct in stroke.

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